Abstract

There is an increasing evidence that Helicobacter pylori may interfere with gastrointestinal metabolism of micronutrients and drugs such as iron, cobalamin, thyroxine and levodopa, with relevant clinical effects. In this review we examine the strength of the causal association and the plausible pathophysiologic mechanisms underlying these adverse effects.

Keywords: Helicobacter pylori, iron, cobalamin, thyroxine, levodopa, malabsorption, gastritis, gastric acidity