Abstract

The physiologic sequelae of Renal Artery Stenosis (RAS) results from narrowing of renal artery lumen which leads to diminished renal perfusion. The consequences of RAS range from asymptomatic to refractory hypertension to ischemic nephropathy eventually leading to end-stage renal disease (ESRD).

Keywords: Renal Artery Stenosis (RAS), hypertension, end-stage renal disease (ESRD), nephropathy, etiology